Wayne Giles

K. H. Jæger, A. G. Edwards, W. R. Giles and A. Tveito

A computational method for identifying an optimal combination of existing drugs to repair the action potentials of SQT1 ventricular myocytes

PLoS Computational Biology

K. H. Jæger, A. G. Edwards, W. R. Giles and A. Tveito

From Millimeters to Micrometers; Re-introducing Myocytes in Models of Cardiac Electrophysiology

Frontiers in Physiology

A. Tveito, K. H. Jæger, M. Maleckar, W. R. Giles and S. Wall

Computational translation of drug effects from animal experiments to human ventricular myocytes

Nature Scientific Reports

J. Koivumäki, R. Clark, D. Belke, C. Kondo, P. Fedak, M. Maleckar and W. R. Giles

Na+ Current Expression in Human Atrial Myofibroblasts: Its Identity and Functional Consequences

Frontiers in Physiology

R. A. Rose, D. Belke, M. Maleckar and W. R. Giles

Ca(2+) Entry Through TRP-C Channels Regulates Fibroblast Biology in Chronic Atrial Fibrillation

Circulation

M. Maleckar, J. L. Greenstein, W. R. Giles and N. A. Trayanova

Electrotonic Coupling Between Human Atrial Myocytes and Fibroblasts Alters Excitability and Repolarization

Biophysical Journal

M. Maleckar, J. L. Greenstein, W. R. Giles and N. A. Trayanova

K+ Current Changes Account for the Rate Dependence of the Action Potential in the Human Atrial Myocyte

American Journal of Physiology - Heart and Circulatory Physiology

M. Maleckar, J. L. Greenstein, N. A. Trayanova and W. R. Giles

Mathematical Simulations of Ligand-Gated and Specific Cell-Type Effects in the Human Atrium

Progress in Biophysics and Molecular Biology

M. Maleckar, J. L. Greenstein, W. R. Giles and N. A. Trayanova

Coupling of Human Atrial Myocytes and Myofibroblasts Can Lead to Conduction Disturbances

HRS Scientific Sessions 2008

S. N. Flaim, W. R. Giles and A. D. McCulloch

Arrhythmogenic Consequences of Na+ Channel Mutations in the Transmurally Heterogeneous Mammalian Left Ventricle: Analysis of the I1768V SCN5A Mutation

Heart Rhythm